It depends on how well you are coping, and your personal take on the medications currently available.http://viartis.net/parkinsons.disease/treatments.htm
The most widely used form of treatment is L-dopa in various forms. L-dopa is transfomed into dopamine in the dopaminergic neurons by L-aromatic amino acid decarboxylase (often known by its former name dopa-decarboxylase). However, only 1-5% of L-DOPA enters the dopaminergic neurons. The remaining L-DOPA is often metabolised to dopamine elsewhere, causing a wide variety of side effects. Due to feedback inhibition, L-dopa results in a reduction in the endogenous formation of L-dopa, and so L-dopa eventually becomes counterproductive
Dopamine agonists are drugs that mimic dopamine by stimulating the dopamine receptors. The dopamine agonists include Bromocriptine (Parlodel), Pramipexole (Mirapex), Ropinirole (Requip), Cabergoline (Dostinex, Cabaser), Lisuride (Revanil), Rotigotine (Neupro) which is applied using a transdermal patch, and Apomorphine hydrochloride (Apokyn) which is administered via injection or infusion. Pergolide (Permax) has been widely withdrawn from use. Besides the side effects they cause, dopamine agonists cause the dopamine receptors to become progressively less sensitive, thereby eventually increasing the symptoms.
MAO-B inhibitors do not directly increase the formation of dopamine or its activity. MAO-B inhibitors instead reduce the symptoms by inhibiting monoamine oxidase-B (MAO-B), which inhibits the breakdown of dopamine secreted by the dopaminergic neurons. The most common MAO-B inhibitors are Selegiline (Eldepryl) and Rasagiline (Azilect). MAO-B inhibitors cause widespread side effects.